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This dysregulation promotes astrocytic ferroptosis, a process that subsequently impairs neuronal viability and function, ultimately contributing to the pathogenesis of DACD (Wang et al., 2024)

Incretin therapies target the class B G protein-coupled receptors GLP-1R and GIPR, expressed mainly in the pancreas and the hypothalamus, while some therapeutical approaches include additional targeting of the related glucagon receptor (GCGR) in the liver