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While GLP-1 therapies work through incretin pathways, cagrilintide activates amylin receptors located in key metabolic control centers of the brain[2]

Such cargos include proteins, siRNA, nanoparticles, oligonucleotides, and other peptides [242]

Compellingly, GCN2 expression in CD4 + T cells is associated with anergy and tolerance to tissue (tumor) damage [151, 152], probably being an interesting molecular target for antagonism, improving antitumor immunity while also restraining tumor cells survival, at least for some tumors

PMID: 26408528 Headley DB et al (2006): Different subcellular distributions of the vesicular monoamine transporter, VMAT2, in subclasses of sympathetic neurons